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In more particular embodiments, the cannabinoid is used to prevent or treat an ischemic or neurodegenerative disease in the central nervous system of a subject, by administering to the subject a therapeutically effective amount of a cannabinoid to protect against oxidative injury to the central nervous system.

The cannabinoid may be any of the compounds set forth above, or more specifically , the compound is CBD monoacetate.

Cannabinoids have been found to have antioxidant properties, unrelated to NMDA receptor antagonism.

This new found property makes cannabinoids useful in the treatment and prophylaxis of wide variety of oxidation associated diseases, such as ischemic, age-related, inflammatory and autoimmune diseases. 1-708, 1991) but cannabidiol is inactive at the NMDA receptor.

Anti-oxidants and anti-inflammatory agents have been proposed to reduce damage, but they often have poor access to structures such as the brain (which are protected by the blood brain barrier). Particular embodiments are dibenzo pyrans and cyclohexenyl benzenediols.

However these endogenous antioxidant systems are often overwhelmed by pathological processes that allow permanent oxidative damage to occur to tissue.

A combination of the injury of hypoxia with the added insult of glutamate toxicity is therefore believed to be ultimately responsible for cellular death. In some particularly disclosed embodiments, the cannabinoid is selected from the group: groups are each independently selected from the groups of hydrogen, lower substituted or unsubstituted alkyl, substituted or unsubstituted carboxyl, substituted or unsubstituted alkoxy, substituted or unsubstituted alcohol, and substituted or unsubstituted ethers, and R where there can be 0 to 3 double bonds on the A ring, as indicated by the optional double bonds indicated by dashed lines on the A ring.

Hence if the additive insult of glutamate toxicity can be alleviated, neurological damage could also be lessened. 5,521,215 (sterospecific ( ) THC enantiomers), and U. The C ring is aromatic, and the B ring can be a pyran.

The invention also includes identifying and administering antioxidant and neuroprotective compounds (such as cannabidiol) which selectively inhibit the enzyme activity of both 5- and 15-lipoxygenase more than the enzyme activity of 12-lipoxygenase.

In addition, such compounds posses low NMDA antagonist activity and low cannabinoid receptor activity.

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Some of the research on these antagonists has focused on cannabinoids, a subset of which have been found to be NMDA receptor antagonists. U. S. Pat. No. This invention provides antioxidant compounds and compositions, such as pharmaceutical compositions, that include cannabinoids that act as free radical. 
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